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Fibular Hemimelia- Drug Today Medical Times

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Various studies have been conducted to ascertain the causes but it is yet to be discovered as to why FH occurs in a newborn. An independent study has revealed that if genes guiding the formation of the limb are activated in an abnormal order, FH can happen.

Researches have highlighted that isolated mutations of genes in the forming limb bud can lead to FH. Though genetic abnormalities are linked to FH, the condition in not heritable. The gene mutations and abnormalities are occurring only in the forming limb and not anywhere else, and thus cannot be transmitted to the next generation.

It is understood that majority of children born with this disorder have no family history of other birth defects. Neither the parents of the child with FH nor the children themselves have any increased risk of producing additional children with this or other birth defects.

Children affected with HF have major complications of limb length discrepancy, foot and ankle deformities and knee deformity.

FH leads to limb length discrepancy because the tibia on the affected side grows at slower rate than the tibia on the opposite side. In addition, many people with FH have slower growing femur as well.

The foot deformity is related to the abnormal ankle joint as well as missing parts of the foot. In patients with fibular hemimelia, the knee joint frequently has a valgus deformity also known knocked knee. This alignment can be related to the lower end of the femur or the upper end of the tibia, or both.

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